Download 2014-2015 Basic and Clinical Science Course (BCSC): Section by American Academy of Ophthalmology, Ramana S. Moorthy MD PDF

By American Academy of Ophthalmology, Ramana S. Moorthy MD

ISBN-10: 161525563X

ISBN-13: 9781615255634

Starts off with an summary of immune-mediated eye affliction, summarizing easy immunologic thoughts, ocular immune responses and specific subject matters in ocular immunology. Discusses the medical method of uveitis and studies noninfectious (autoimmune) and infectious types of uveitis, with an extended part on viral uveitis and new fabric on infectious and noninfectious scleritis.

Enhanced detection of infectious brokers by way of immunologic and genetic equipment and biologic therapeutics are specific. additionally covers endophthalmitis, masquerade syndromes, issues of uveitis and ocular elements of AIDS.

Upon of completion of part nine, readers might be capable to:

Identify common and particular pathophysiologic strategies that impact the constitution and serve as of the uvea, lens, intraocular cavities, retina, and different tissues in acute and persistent intraocular inflammation
Differentiate and establish infectious and noninfectious uveitic entities
Choose applicable exam suggestions and appropriate ancillary stories in line with no matter if an infectious or noninfectious reason is suspected

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Additional resources for 2014-2015 Basic and Clinical Science Course (BCSC): Section 9: Intraocular Inflammation and Uvetis

Example text

In recent years, 2 additional T-cell subsets have been recognized. Thl7 cells are so-named because they produce IL-17, as well as IL-21 and -22. Thl7 cells have been implicated in a number of autoimmune conditions, including some forms of uveitis. T regulatory (Treg) cells form another subset of helper T cells; they are identified not by their cytokine profile but by the simultaneous surface expression of CD4, CD25, and Foxp3. Treg cells are essentially suppressor-type T cells that down-regulate other T cell populations.

Modified with permission from Pepose JS, Holland GN, Wi/helmus KR, eds Ocular Infection and Immunity. St Louis, MO: Mosbv: 1996) inhibitors does diminish the onset of mild CME after cataract surgery, and both topical and systemic nonsteroidal anti-inflammatory drug (NSAID) therapy might be efficacious in the treatment of persistent CME. Systemic NSAID therapy is effective in treating recurrent episcleritis and mild cases of scleritis. Prostaglandins and other eicosanoids indisputably play a major role in the physiology of the eye, reaching far beyond their putative participation as mediators of inflammation.

Phagocytes are endowed with multiple means of destroying microorganisms, especially antimicrobial polypeptides that reside within cytoplasmic granules, reactive oxygen radicals generated from oxygen during the respiratory burst, and reactive nitrogen radicals. Although these mechanisms are primarily designed to destroy pathogens, released contents such as lysosomal enzymes may contribute to the amplification of inflammation and tissue damage. Innate Mechanisms for the Recruitment and Activation of Macrophages Monocyte-derived macrophages are the second important type of effector cell for the in nate immune response following trauma or acute infection.

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